Gene information
Official symbol SAMSN1
Official full name SAM domain, SH3 domain and nuclear localization signals 1
Gene type protein-coding gene
Chromosome21q11.2
Alias symbolNASH1; SASH2; SH3D6B; HACS1; SLy2
Gene familySAM and SH3 domain containing; Sterile alpha motif domain containing
Resources
Entrez Gene 64092
HGNC HGNC:10528
Ensembl ENSG00000155307
Vega OTTHUMG00000074317
UCSC uc002yju.3
GO terms: 14 records
GO Description Category LogP Number of genes Detail
GO:0001933 negative regulation of protein phosphorylation GO Biological Processes -19.8214973 59 More
GO:0002683 negative regulation of immune system process GO Biological Processes -26.6415063 77 More
GO:0002695 negative regulation of leukocyte activation GO Biological Processes -9.84552767 31 More
GO:0010563 negative regulation of phosphorus metabolic process GO Biological Processes -21.7837721 71 More
GO:0031252 cell leading edge GO Cellular Components -1.97716247 28 More
GO:0031400 negative regulation of protein modification process GO Biological Processes -17.0986645 70 More
GO:0042326 negative regulation of phosphorylation GO Biological Processes -20.6187298 64 More
GO:0045936 negative regulation of phosphate metabolic process GO Biological Processes -21.8424865 71 More
GO:0050730 regulation of peptidyl-tyrosine phosphorylation GO Biological Processes -27.1120967 60 More
GO:0050732 negative regulation of peptidyl-tyrosine phosphorylation GO Biological Processes -3.13397845 4 More
GO:0050777 negative regulation of immune response GO Biological Processes -11.1440698 32 More
GO:0050866 negative regulation of cell activation GO Biological Processes -13.9027363 39 More
GO:0050869 negative regulation of B cell activation GO Biological Processes -2.50832748 6 More
GO:0051250 negative regulation of lymphocyte activation GO Biological Processes -7.76846228 25 More
Related study: 1 record
Molecular factor Omics type Application Objective MI type Reference
SAMSN1 transcriptomics acute ST elevation myocardial infarction[diagnosis] acute ST elevation myocardial infarction Gobbi G et al. Sci Rep. 2019
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